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Explaining Heartburn-Acid Reflux Disorder

 

 

Acid reflux disease and heartburn are frequently used terms for symptoms of GERD, or gastroesophageal reflux disease. This is a chronic condition wherein the digestive fluids in the stomach rise back into the esophagus. If ignored, this condition may lead to esophageal damage which could require surgical intervention or become life-threatening. To halt complications from acid reflux, once medical treatment for acid reflux disease has been started, it is often or usually necessary to continue treatment indefinitely. Medications can help to limit the damage but, if discontinued, the disorder and its damage may reoccur.

Specifically, there is a spot as the esophagus passes through the diaphragm where the lower end of the esophagus joins the stomach and this junction is controlled by a one-way valve called the, "lower esophageal sphincter," or LES. When this structure does not shut completely after swallowing, or is pushed open, liquid stomach contents can rise back upward into the esophagus.

There is no single best way to resolve this, but several of the major causes are well understood and, luckily, the majority of them are things which can be controlled to at least some degree. Pressure arising from the stomach and its contents can push on the LES permitting acid to leak back through. Being overweight and eating large quantities are two things which increase this pressure.

Acidic substances can also damage the function of the LES, whether the acid is in the food itself or is produced by the stomach to digest the food. Large, rich or high fat substances take longer to digest and take more acid for digestion, so they can actually cause trouble in two ways.

One of the simplest, least restrictive methods of improving acid reflux disease is merely to eat your normal diet over the day in many small meals, as opposed to three larger ones. This lifestyle change has also helped some people to lose weight since they don't become ravenous and overeat before feeling satisfied.

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